Given that NIDCD’s mission is to “conduct and support research” into “hearing, balance, smell, taste, voice, speech and language" and that its Chemical Senses Branch funded the first studies identifying CO as a "secondary neuromessenger" (aka a neurotransmitter that modulates sensory nerve thresholds) in the early 1990s ...
Why has NIDCD continued to focus almost exclusively on disorders involving the partial or complete loss of sensory function (deafness, blindness, anosmia, agusia, etc) and not supported more research into disorders involving sensory hypersensitivities, either alone--such as photophobia and phonophobia--or in combination, such as multi-sensory sensitivity or MUSES syndrome, which is the hallmark of chronic CO poisoning?
